Hot Sale Skf-525a Hcl, Cytochrome P450 Inhibitor (tbi4050) - Today Only [BLTXQiS4]

SKF-525A HCl: A Potent Cytochrome P450 Inhibitor

SKF-525A HCl, also known as Proadifen, is a highly effective inhibitor of the cytochrome P450 enzyme system. This compound is widely recognized for its ability to block the acetylcholine receptor and inhibit nitric oxide synthase, making it a valuable tool in various research applications. With a purity of greater than 98%, SKF-525A HCl offers reliable and consistent performance. Its broad-spectrum inhibitory properties make it suitable for studying the role of cytochrome P450 enzymes in various biological processes.

• High purity (>98%) ensures consistent results.
• Broad-spectrum cytochrome P450 monoxygenase inhibitor.

Product Specifications and Technical Details

SKF-525A HCl is available as a white powder, highly soluble in DMSO (up to 20 mg/ml). This solubility allows for easy preparation of solutions that can be stored at -20°C for up to 3 months without degradation. The molecular formula of SKF-525A HCl is C₂₃H₃₁NO₂ ∙ HCl, with a formula weight of 390.00. The CAS number for this compound is 62-68-0. These specifications make SKF-525A HCl a versatile and reliable choice for researchers in need of a potent cytochrome P450 inhibitor.

• Formal Name: N,N-Diethylaminoethyl-2,2-diphenylvalerate hydrochloride.
• Storage: -20°C for stability and long-term storage.

Applications and Benefits of SKF-525A HCl

SKF-525A HCl is a versatile research tool with a wide range of applications. Its ability to inhibit cytochrome P450 enzymes, block acetylcholine receptors, and inhibit nitric oxide synthase makes it ideal for studying inflammation, neural excitability, and various metabolic pathways. This compound is particularly useful in Müller glial cell research, where it increases inflammatory cytokine expression, and in studies involving rat 5-HT neurons, where it inhibits excitability. With its ease of use and long shelf life, SKF-525A HCl is a must-have for any laboratory focused on neuropharmacology, inflammation, and metabolic research.

• Inhibits nitric oxide synthase and glibenclamide-sensitive K+ channels.
• Increases inflammatory cytokine expression in Müller glial cells.