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Fast Shipping Brilliant Violet 711™ Anti-mouse/human Klrg1 (mafa) Series [n65w8a1D]

$56.99 $182.99 -69%

Brilliant Violet 711™ Anti-Mouse/Human KLRG1 (MAFA) The Fast Shipping Brilliant Violet 711™ Anti-mouse/human Klrg1 (mafa) Series [n65w8a1D] antibody is a powerful tool for researchers studying immune cell function and regulation. This monoclonal antibody, clone 2F1/KLRG1, offers ex

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Brilliant Violet 711™ Anti-Mouse/Human KLRG1 (MAFA)

The Fast Shipping Brilliant Violet 711™ Anti-mouse/human Klrg1 (mafa) Series [n65w8a1D] antibody is a powerful tool for researchers studying immune cell function and regulation. This monoclonal antibody, clone 2F1/KLRG1, offers exceptional specificity and reactivity across both mouse and human samples. With its BV711 conjugation, this antibody provides high sensitivity and excellent fluorescence properties for flow cytometry analysis.

Product Features and Specifications

  • Clone: 2F1/KLRG1
  • Antibody Type: Monoclonal
  • Reactivity: Mouse, Human
  • Format: BV711
  • Host: Syrian Hamster
  • Isotype: IgG
This antibody is highly versatile and suitable for a variety of applications, including flow cytometry. Its BV711 conjugation ensures bright, stable fluorescence, making it ideal for multi-color experiments and complex staining panels.

Practical Applications and Benefits

The Fast Shipping Brilliant Violet 711™ Anti-mouse/human Klrg1 (mafa) Series [n65w8a1D] antibody is particularly valuable for investigating the role of KLRG1 in immune cell subsets, such as NK cells, LAK cells, and T cells. KLRG1 has been shown to regulate NK cell homeostasis and modulate immune responses by interacting with cadherins. This antibody allows researchers to easily identify and characterize KLRG1-expressing cells, facilitating a deeper understanding of immune system function. With its robust performance and convenient handling, this antibody is a must-have for any laboratory engaged in immunological research.

What Our Customers Say

January 5, 2026

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January 5, 2026

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January 5, 2026

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- Fitzwilliam C..

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