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Multi-purpose Recombinant Mouse Cxcl2/mip-2 Protein Massive 45% Savings [Oj3Nqr0x]

$72.99 $233.99 -69%

Multi-purpose Recombinant Mouse Cxcl2/mip-2 Protein Massive 45% Savings [Oj3Nqr0x] Discover the power of recombinant mouse CXCL2/MIP-2 protein, a key player in immune response and inflammation research. This high-quality protein, produced through recombinant technology, offers exceptional purit

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Multi-purpose Recombinant Mouse Cxcl2/mip-2 Protein Massive 45% Savings [Oj3Nqr0x]

Discover the power of recombinant mouse CXCL2/MIP-2 protein, a key player in immune response and inflammation research. This high-quality protein, produced through recombinant technology, offers exceptional purity and bioactivity, making it an ideal tool for various applications in the laboratory.

Product Features

Our recombinant mouse CXCL2/MIP-2 protein is expressed in E. coli and purified to a purity of >98% as determined by reducing SDS-PAGE. It carries an N-His tag and exhibits strong bioactivity, with an ED50 of <0.5 ng/mL when measured by its ability to chemoattract BaF3 cells transfected with human CXCR2. Key specifications include a calculated molecular weight of 8.7 kDa and an observed molecular weight of 11 kDa.
  • High purity and bioactivity ensure reliable results in your experiments.
  • Expressed in E. coli for easy production and purification.

Applications and Benefits

The recombinant mouse CXCL2/MIP-2 protein is a versatile tool for studying inflammation, immune response, and cell signaling. Its ability to chemoattract cells and its association with neutrophil influx in pulmonary inflammation and glomerulonephritis make it particularly valuable for researchers investigating inflammatory diseases. This protein is suitable for use in various applications, including:
  • Cell-based assays to study chemotaxis and immune cell interactions.
  • Development of therapeutic agents targeting inflammatory diseases.
  • Understanding the molecular mechanisms of inflammation and immune response.
  • What Our Customers Say

    January 11, 2026

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    January 11, 2026

    Absolutely no complaints!

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    January 11, 2026

    Absolutely no complaints!

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